Recently, some scholars have confirmed that hyperbaric oxygen therapy combined with weight-bearing has achieved satisfactory results in animal models of avascular necrosis of the femoral head. Recently, Reis reported 12 patients with stage I ischemic necrosis of the femoral head (of which 4 cases were bilateral necrosis), who underwent hyperbaric oxygen therapy every day for 100 consecutive days, 81 patients had normal MRI examination, while the control group had only 17％ recovery rate. He believes that for patients with avascular necrosis of the femoral head, hyperbaric oxygen therapy is effective.

　　Hyperbaric oxygen treatment mechanism: ① increase Arterial blood oxygen partial pressure, increase capillary oxygen diffusion distance, increase local oxygen partial pressure, reduce acid metabolite aggregation; ② enhance the proliferation and division of osteoblasts, osteoclasts, fibroblasts, endothelial cells, accelerate granulation tissue, The proliferation of fibrous tissue and connective tissue accelerates the growth of bone tissue and accelerates the repair of necrotic bone tissue; ③ enhances the ability of phagocytic cells to engulf bacteria and necrotic tissue, and enhances the ability to resist infection and clear the lesion. In addition, the following methods can also be used:

　　 1. High-energy shock wave therapy: The application of high-energy shock wave therapy in orthopedics is not to lyse tissue, but to induce the formation of blood vessels in the tissue, which has slight trauma, little pain, and cost Low characteristics. Some scholars believe that it causes cavitation of tissue by a certain amount of ultrasonic pressure, resulting in the local formation of micro-fractures and fine hematomas, which in turn induces vascular growth and osteogenesis; some scholars have confirmed through animal experiments that its mechanism of action is mainly to activate angiogenesis and growth Factor expression, angiogenesis and growth factors include: endothelial nitric oxide synthase (endothelial nitric oxide synthase (eNOS), vascular endothelial growth factor (vessel endothelial growth factor, VEGF) and proliferating cell nuclear antigen Antigen (PCNA) is currently believed to be related to stimulating the growth of new blood vessels and increasing blood supply to promote bone tissue regeneration.

　　 2. Electromagnetic field treatment: The principle of electrical stimulation to treat femoral head necrosis is that it can promote the formation of new blood vessels, inhibit the absorption of dead bones, improve the differentiation ability of osteoblasts, and promote the repair of bone damage. Steinberg et al. inserted the electrode into the central decompression hole for direct current stimulation, while implanting cancellous bone to treat 18 femoral head necrosis. The average follow-up was 5 months. There was no increase in pain in the electrical stimulation group and 14 cases of pain relief.

　　3. Cytokine therapy: With the progress of research on osteoclast regulation, recently, foreign scholars have obtained a secreted glycoprotein through molecular cloning technology, named: protective protein/broken Osteoclastogenesis inhibitory factor inhibitory factor, OPG/OCIF). In this regard, many scholars have conducted a large number of studies to explain that OPG/OCIF has the physiological function of inhibiting osteoclast differentiation and activity. It is believed that in the early stage of hormonal osteonecrosis, if the concentration of OPG/OCIF in the bone is increased, it can inhibit bone Mass loss provides a new way for the early treatment of hormonal femoral head necrosis.

　　 4. Autologous bone marrow cell transplantation: In the latest report, Gangji V described the use of autologous bone marrow mononuclear cell transplantation for the treatment of adult stage Ⅰ-Ⅱ femoral head necrosis, and considered it to be a safe and effective treatment. However, he also admitted that the number of cases is still small and the follow-up time is limited. The feasibility needs further study.

　　 In addition, the motherland medicine believes that hormones belong to the category of external evils, "necrosis" is caused by stagnation of blood and blood stasis, blood circulation disorders belong to "stasis", and hyperlipidemia is a blood stasis syndrome, which is "dirty" blood". Hyperviscosity, including hematocrit, whole blood viscosity, plasma viscosity, etc., is higher than normal, indicating that the blood is in a highly concentrated, viscous, and aggregated state, and there is a "blood disorder", similar to "inner node is blood stasis". Recent studies have shown that TXA2-PGI2 imbalance and vascular endothelial cell function changes are qualitatively related to blood stasis. When the function of vascular endothelial cells is inhibited, the synthesis of PGI2 is reduced, and/or TXA2 is excessively produced, the ratio of TXA2/PGI2 is increased, and the fibrinolysis system and platelet function are disordered, resulting in "blood loss" and "blood stasis. Blood stasis. Because hormones can cause hyperlipidemia, hyperviscosity, and TXA2-PGI2 imbalance, it is generally believed that the key cause of hormonal femoral head necrosis is blood stasis, and the main method in the clinic is promoting blood circulation and removing blood stasis.